Please use this identifier to cite or link to this item: http://hdl.handle.net/11434/929
Full metadata record
DC FieldValueLanguage
dc.contributor.authorWilliams, Gavin-
dc.contributor.authorBanky, Megan-
dc.contributor.authorMcKenzie, Dean-
dc.contributor.authorOlver, John-
dc.date2016-11-
dc.date.accessioned2016-11-28T05:21:25Z-
dc.date.available2016-11-28T05:21:25Z-
dc.date.issued2016-
dc.identifier.citationBrain Inj. 2017;31(2):193-198-
dc.identifier.issn0269-9052en_US
dc.identifier.issn1362-301Xen_US
dc.identifier.urihttp://hdl.handle.net/11434/929-
dc.description.abstractPRIMARY OBJECTIVES: The main aim of this study was to determine whether the presence of distal lower-limb spasticity had a greater impact on mobility for those who had greater levels of muscle paresis following traumatic brain injury (TBI). RESEARCH DESIGN: This was a cross-sectional cohort study of convenience. Seventy-five people attending physiotherapy for mobility limitations following TBI participated in this study. All participants had sustained a moderate-severe TBI and were grouped according to the presence or absence of ankle plantarflexor spasticity for comparison. MAIN OUTCOMES AND RESULTS: The primary outcome measure for mobility was self-selected walking speed and the primary outcome measure for muscle strength was hand-held dynamometry. Secondary outcome measures for mobility and muscle strength were the High-level Mobility Assessment Tool (HiMAT) and ankle power generation (APG) at push-off. Spasticity was quantified with the Modified Tardieu scale. Participants with ankle plantarflexor spasticity (Group 2) had slower self-selected walking speeds. There was no statistically significant effect for Group and plantarflexor strength (p = 0.81). CONCLUSION: Although participants with ankle plantarflexor spasticity walked significantly slower than those without, the presence of ankle plantarflexor spasticity did not lead to greater mobility limitations for those who were weak.en_US
dc.publisherTaylor & Francis Onlineen_US
dc.subjectMuscle Weaknessen_US
dc.subjectMobility Limitationen_US
dc.subjectAnkleen_US
dc.subjectAnkle Jointen_US
dc.subjectAnkle Injuriesen_US
dc.subjectInjuries, Ankleen_US
dc.subjectRange of Motion, Articularen_US
dc.subjectBrain Injuriesen_US
dc.subjectTrauma, Brainen_US
dc.subjectInjuries, Brainen_US
dc.subjectTBIen_US
dc.subjectTraumatic Brain Injuryen_US
dc.subjectWalkingen_US
dc.subjectGaiten_US
dc.subjectAmbulationen_US
dc.subjectMuscle Strengthen_US
dc.subjectLocomotionen_US
dc.subjectMovementen_US
dc.subjectMotionen_US
dc.subjectRehabilitationen_US
dc.subjectRecovery of Functionen_US
dc.subjectPhysiotherapyen_US
dc.subjectMuscle Spasticityen_US
dc.subjectHiMATen_US
dc.subjectHigh-level Mobility Assessment Toolen_US
dc.subjectModified Tardieu Scaleen_US
dc.subjectEpworth Monash Rehabilitation Medicine Unit, Victoria, Australia.en_US
dc.subjectNeurosciences Clinical Institute, Epworth HealthCare, Victoria, Australiaen_US
dc.subjectRehabilitation, Mental Health and Chronic Pain Clinical Institute, Epworth HealthCare, Victoria, Australiaen_US
dc.titleAnkle plantarflexor spasticity is not differentially disabling for those who are weak following traumatic brain injury.en_US
dc.typeJournal Articleen_US
dc.identifier.doi10.1080/02699052.2016.1218548en_US
dc.identifier.journaltitleBrain Injuryen_US
dc.description.pubmedurihttps://www.ncbi.nlm.nih.gov/pubmed/27880057en_US
dc.description.affiliatesThe University of Melbourne, Melbourne, Victoria, Australia.en_US
dc.description.affiliatesSchool of Nursing and Midwifery, Deakin University, Melbourne, Victoria, Australia.en_US
dc.type.studyortrialCohort Studyen_US
dc.type.contenttypeTexten_US
Appears in Collections:Musculoskeletal
Neurosciences
Rehabilitation

Files in This Item:
There are no files associated with this item.


Items in Epworth are protected by copyright, with all rights reserved, unless otherwise indicated.